1025 Walnut Street
Suite 311
Philadelphia, PA 19107
(215) 503-9428
(215) 503-5731 fax
Suite 311
Philadelphia, PA 19107
(215) 503-9428
(215) 503-5731 fax
Most Recent Peer-reviewed Publications
- Tumor necrosis factor receptor-2 signaling attenuates vein graft neointima formation by promoting endothelial recovery
- Expression of tumor necrosis factor receptor-1 in arterial wall cells promotes atherosclerosis
- Activation of vascular smooth muscle cells by TNF and PDGF: Overlapping and complementary signal transduction mechanisms
- Vein graft neointimal hyperplasia is exacerbated by tumor necrosis factor receptor-1 signaling in graft-intrinsic cells
- Graft-Extrinsic Cells Predominate in Vein Graft Arterialization
- Phosphorylation of the platelet-derived growth factor receptor-β and epidermal growth factor receptor by G protein-coupled receptor kinase-2. Mechanisms for selectivity of desensitization
- Overexpression of G protein-coupled receptor kinase-2 in smooth muscle cells reduces neointimal hyperplasia
- Overexpression of G protein - Coupled receptor kinase-2 in smooth muscle cells attenuates mitogenic signaling via G protein - Coupled and platelet-derived growth factor receptors
- G protein-coupled receptor kinase 3 (GRK3) gene disruption leads to loss of odorant receptor desensitization
- A tumor necrosis factor (TNF) receptor-IgG heavy chain chimeric protein as a bivalent antagonist of TNF activity