Kimmel Cancer Center
233 S. Tenth Street
Suite 1035
Philadelphia, PA 19107
(215) 503-9350
(215) 923-4498 fax
233 S. Tenth Street
Suite 1035
Philadelphia, PA 19107
(215) 503-9350
(215) 923-4498 fax
Most Recent Peer-reviewed Publications
- Erratum: ChIP sequencing of cyclin D1 reveals a transcriptional role in chromosomal instability in mice (J Clin Invest. (2013) 123:5 (2332) 10.1172/JCI70042)
- Novel oncogene-induced metastatic prostate cancer cell lines define human prostate cancer progression signatures
- Mitochondrial fission induces glycolytic reprogramming in cancer-associated myofibroblasts, driving stromal lactate production, and early tumor growth
- Cyclin D1 induces chromosomal instability
- ChIP sequencing of cyclin D1 reveals a transcriptional role in chromosomal instability in mice
- Mammary gland selective excision of c-Jun identifies its role in mRNA splicing
- Caveolin-1 overexpression enhances androgen-dependent growth and proliferation in the mouse prostate
- Examining the role of cyclin D1 in breast cancer
- Disruption of a Sirt1-dependent autophagy checkpoint in the prostate results in prostatic intraepithelial neoplasia lesion formation
- Cell fate determination factor Dachshund reprograms breast cancer stem cell function
- The canonical NF-κB pathway governs mammary tumorigenesis in transgenic mice and tumor stem cell expansion
- C-jun inhibits mammary apoptosis in vivo
- The autophagic tumor stroma model of cancer: Role of oxidative stress and ketone production in fueling tumor cell metabolism
- HIF1-alpha functions as a tumor promoter in cancer associated fibroblasts, and as a tumor suppressor in breast cancer cells: Autophagy drives compartment-specific oncogenesis
- Loss of stromal caveolin-1 leads to oxidative stress, mimics hypoxia and drives inflammation in the tumor microenvironment, conferring the "reverse Warburg effect": A transcriptional informatics analysis with validation
- Attenuation of Forkhead signaling by the retinal determination factor DACH1
- Transcriptional evidence for the "Reverse Warburg Effect" in human breast cancer tumor stroma and metastasis: Similarities with oxidative stress, inflammation, Alzheimer's disease, and "Neuron-Glia Metabolic Coupling"
- Activating peroxisome proliferator-activated receptor γ mutant promotes tumor growth in vivo by enhancing angiogenesis
- The reverse Warburg effect: Aerobic glycolysis in cancer associated fibroblasts and the tumor stroma
- p21CIP1 attenuates Ras- and c-Myc-dependent breast tumor epithelial mesenchymal transition and cancer stem cell-like gene expression in vivo