233 South Tenth Street
Bluemle Life Sciences Building, Room 531
Philadelphia, PA 19107-5541
(215) 503-4634
(215) 503-5393 fax
Bluemle Life Sciences Building, Room 531
Philadelphia, PA 19107-5541
(215) 503-4634
(215) 503-5393 fax
Most Recent Peer-reviewed Publications
- Interspecies protein substitution to investigate the role of the lyssavirus glycoprotein
- A replication-deficient rabies virus vaccine expressing Ebola virus glycoprotein is highly attenuated for neurovirulence
- Immune Clearance of Attenuated Rabies Virus Results in Neuronal Survival with Altered Gene Expression
- Further characterization of the immune response in mice to inactivated and live rabies vaccines expressing Ebola virus glycoprotein
- Targeted single-neuron infection with rabies virus for transneuronal multisynaptic tracing
- The role of micronutrients in the diet of HIV-1-infected individuals
- A role for granulocyte-macrophage colony-stimulating factor in the regulation of CD8+ T cell responses to rabies virus
- Proliferating cell nuclear antigen is required for loading of the SMCX/KMD5C histone demethylase onto chromatin
- Inactivated or live-attenuated bivalent vaccines that confer protection against rabies and Ebola viruses
- MicroRNA-122: a therapeutic target for hepatitis C virus (HCV) infection.
- Immunization of mice with the non-toxic HC50 domain of botulinum neurotoxin presented by rabies virus particles induces a strong immune response affording protection against high-dose botulinum neurotoxin challenge
- Rabies Virus as a Research Tool and Viral Vaccine Vector
- Rabies virus (RV) glycoprotein expression levels are not critical for pathogenicity of RV
- Dendritic cells infected by recombinant rabies virus vaccine vector expressing HIV-1 Gag are immunogenic even in the presence of vector-specific immunity
- Rabies virus infection induces type I interferon production in an IPS-1 dependent manner while dendritic cell activation relies on IFNAR signaling.
- Induction of neutralizing antibody responses to anthrax protective antigen by using influenza virus vectors: Implications for disparate immune system priming pathways
- Rabies virus infection induces type I interferon production in an IPS-1 dependent manner while dendritic cell activation relies on IFNAR signaling
- Characterization of a single-cycle rabies virus-based vaccine vector
- A novel composite immunotoxin that suppresses rabies virus production by the infected cells
- Attenuation of rabies virulen Takeover by the cytoplasmic domain of its envelope protein