900 Walnut Street
Suite 200
Philadelphia, PA 19107
(215) 955-7952
(215) 955-9976 fax
Suite 200
Philadelphia, PA 19107
(215) 955-7952
(215) 955-9976 fax
Most Recent Peer-reviewed Publications
- Stiff person syndrome improvement with chemotherapy in a patient with cutaneous t cell lymphoma
- Pearls and oy-sters: Neurosyphilis presenting as mesial temporal encephalitis
- Reply
- HIV infection and antiretroviral therapy have divergent effects on mitochondria in adipose tissue
- Autoimmune stiff person syndrome and related myelopathies: Understanding of electrophysiological and immunological processes
- Rituximab induces sustained reduction of pathogenic B cells in patients with peripheral nervous system autoimmunity
- Atypical clinically diagnosed stiff-person syndrome response to dantrolene-a refractory case
- Reply: Comment on alemtuzumab and inclusion body myositis
- Effect of Alemtuzumab (CAMPATH 1-H) in patients with inclusion-body myositis
- Placebo-controlled trial of rituximab in IgM anti-myelin-associated glycoprotein antibody demyelinating neuropathy
- T cell receptor profiling in muscle and blood lymphocytes in sporadic inclusion body myositis
- Inclusion body myositis with human immunodeficiency virus infection: Four cases with clonal expansion of viral-specific T cells
- Intravenous immune globulin in hereditary inclusion body myopathy: A pilot study
- Autoimmunity to GABAA-receptor-associated protein in stiff-person syndrome
- Stiff person syndrome with cerebellar disease and high-titer anti-GAD antibodies
- A neuropsychological assessment of phobias in patients with stiff person syndrome
- Bilateral pallidotomy for severe dystonia in an 18-month-old child with glutaric aciduria
- Anti-glutamic acid decarboxylase antibodies in the serum and cerebrospinal fluid of patients with stiff-person syndrome: Correlation with clinical severity
- Upregulated inducible co-stimulator (ICOS) and ICOS-ligand in inclusion body myositis muscle: Significance for CD8+ T cell cytotoxicity
- Hypoglycosylation of α-dystroglycan in patients with hereditary IBM due to GNE mutations