900 Walnut Street
Suite 200
Philadelphia, PA 19107
(215) 955-1234
(215) 955-1390 fax
Suite 200
Philadelphia, PA 19107
(215) 955-1234
(215) 955-1390 fax
Most Recent Peer-reviewed Publications
- Immune tolerance induced by intravenous transfer of immature dendritic cells via up-regulating numbers of suppressive IL-10+ IFN-γ+-producing CD4+ T cells
- Intravenous transfer of apoptotic cell-treated dendritic cells leads to immune tolerance by blocking Th17 cell activity
- Role of Th17 cells in the pathogenesis of CNS inflammatory demyelination
- Generation of large numbers of highly purified dendritic cells from bone marrow progenitor cells after co-culture with syngeneic murine splenocytes
- Independent and interdependent immunoregulatory effects of IL-27, IFN-β, and IL-10 in the suppression of human Th17 cells and murine experimental autoimmune encephalomyelitis
- Low dose zymosan ameliorates both chronic and relapsing experimental autoimmune encephalomyelitis
- Role of serine proteases in inflammation: Bowman-Birk protease inhibitor (BBI) as a potential therapy for autoimmune diseases
- Accelerated and enhanced effect of CCR5-transduced bone marrow neural stem cells on autoimmune encephalomyelitis
- TCR stimulation upregulates MS4a4B expression through induction of AP-1 transcription factor during T cell activation
- CNS-specific therapy for ongoing EAE by silencing IL-17 pathway in astrocytes
- Intravenous tolerance effectively overcomes enhanced pro-inflammatory responses and experimental autoimmune encephalomyelitis severity in the absence of IL-12 receptor signaling
- IL-10 deficiency blocks the ability of LPS to regulate expression of tolerance-related molecules on dendritic cells
- Interleukin-10 plays a crucial role in suppression of experimental autoimmune encephalomyelitis by Bowman-Birk inhibitor
- Bowman-Birk inhibitor attenuates experimental autoimmune encephalomyelitis by delaying infiltration of inflammatory cells into the CNS
- Generation of immunogenic and tolerogenic clinical-grade dendritic cells
- IL-9 is important for T-cell activation and differentiation in autoimmune inflammation of the central nervous system
- Kit (W-sh) mice develop earlier and more severe experimental autoimmune encephalomyelitis due to absence of immune suppression
- The encephalitogenicity of TH 17 cells is dependent on IL-1- and IL-23-induced production of the cytokine GM-CSF
- 3G11 expression in CD4+ T cell-mediated autoimmunity and immune tolerance
- Oral resveratrol reduces neuronal damage in a model of multiple sclerosis