16D0 Pestell, Richard G. - Thomas Jefferson University - Thomas Jefferson University
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Richard G. Pestell, MD, PhD

Contact Dr. Pestell

233 South 10th Street
Room 1050
Philadelphia, PA 19107

(215) 503-5692
(215) 503-9334 fax

Most Recent Peer-reviewed Publications

  1. Antibiotics for cancer therapy
  2. Kinase-independent role of cyclin D1 in chromosomal instability and mammary tumorigenesis
  3. BCL-2 family protein, BAD is down-regulated in breast cancer and inhibits cell invasion
  4. Targeting tumor-initiating cells: Eliminating anabolic cancer stem cells with inhibitors of protein synthesis or by mimicking caloric restriction
  5. Loss of sirt1 promotes prostatic intraepithelial neoplasia, reduces mitophagy, and delays park2 translocation to mitochondria
  6. Sirt1-deficient mice have hypogonadotropic hypogonadism due to defective GnRH neuronal migration
  7. A community effort to assess and improve drug sensitivity prediction algorithms
  8. A community computational challenge to predict the activity of pairs of compounds
  9. CCR5 receptor antagonists block metastasis to bone of v-Src oncogene-transformed metastatic prostate cancer cell lines
  10. 05B4
  11. Cyclin D1 integrates estrogen-mediated DNA damage repair signaling
  12. CAPER, a novel regulator of human breast cancer progression
  13. Role of Cancer Stem Cells in Metastasis
  14. Overview of cyclins D1 function in cancer and the CDK inhibitor landscape: Past and present
  15. Long and noncoding RNAs (lnc-RNAs) determine androgen receptor dependent gene expression in prostate cancer growth in vivo
  16. Cell fate factor DACH1 represses YB-1-mediated oncogenic transcription and translation
  17. Identification of a cyclin D1 network in prostate cancer that antagonizes epithelial-mesenchymal restraint
  18. MicroRNA-mediated cancer metastasi 0612 s regulation via heterotypic signals in the microenvironment
  19. Acetylation-defective mutants of PparĪ³ are associated with decreased lipid synthesis in breast cancer cells
  20. The metastatic potential of triple-negative breast cancer is decreased via caloric restriction-mediated reduction of the miR-17~92 cluster
  21. miR-17/20 sensitization of breast cancer cells to chemotherapy-induced apoptosis requires Akt1
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