0B68 Benovic, Jeffrey L. - Thomas Jefferson University - Thomas Jefferson University
jlb104

Jeffrey L. Benovic, PhD

Contact Dr. Benovic

233 S. 10th Street
Philadelphia, PA 19107

(215) 503-4607
(215) 503-5393 fax

Most Recent Peer-reviewed Publications

  1. Structural biology: Arresting developments in receptor signalling
  2. New frontiers in kinases
  3. Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization
  4. Consequence of the tumor-associated conversion to cyclin D1b
  5. Development and characterization of pepducins as Gs-biased allosteric agonists
  6. Role of β-arrestins and arrestin domain-containing proteins in G protein-coupled receptor trafficking
  7. Call for papers! a special thematic compilation/special issue crossover with ACS chemical biology, ACS medicinal chemistry letters, and the journal of medicinal chemistry focused on new frontiers in kinases
  8. Caspase-cleaved arrestin-2 and BID cooperatively facilitate cytochrome C release and cell death
  9. Editorial overview: Cell regulation: The ins and outs of G protein-coupled receptors
  10. β-Arrestins and G protein-coupled receptor trafficking
  11. Pepducin targeting the C-X-C chemokine receptor type 4 acts as a biased agonist favoring activation of the inhibitory G protein
  12. G protein-coupled receptor kinase 2 (GRK2) is localized to centrosomes and mediates epidermal growth factor-promoted centrosomal separation
  13. β-Arrestins and G protein-coupled receptor trafficking
  14. Identification of phosphorylation sites in the COOH-terminal tail of the μ-opioid receptor
  15. G-protein-coupled receptors signal victory
  16. Identification and characterization of distinct c-terminal domains of the human hydroxycarboxylic acid receptor-2 that are essential for receptor export, constitutive activity, desensitization, and internalization
  17. G protein-coupled receptor kinase 5 phosphorylates nucleophosmin and regulates cell sensitivity to polo-like kinase 1 inhibition
  18. Structural domains required for Caenorhabditis elegans G protein-coupled receptor kinase 2 (GRK-2) function in Vivo
  19. G protein-coupled receptor kinase 5 is localized to centrosomes and regulates cell cycle progression
  20. Suppression of G-protein-coupled receptor kinase 3 expression is a feature of classical GBM that is required for maximal growth
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