0B68 Merry, Diane E - Thomas Jefferson University - Thomas Jefferson University
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Diane E. Merry, PhD

Contact Dr. Merry

233 South 10th Street
228 BLSB
Philadelphia, PA 19107

(215) 503-4907
(215) 923-9162 fax

Most Recent Peer-reviewed Publications

  1. Behavioral phenotypes in males with XYY and possible role of increased NLGN4Y expression in autism features
  2. Transcriptional activation of TFEB/ZKSCAN3 target genes underlies enhanced autophagy in spinobulbar muscular atrophy
  3. Cell biological approaches to investigate polyglutamine-expanded AR metabolism
  4. Activation of Hsp70 reduces neurotoxicity by promoting polyglutamine protein degradation
  5. A polyglutamine expansion disease protein sequesters PTIP to attenuate DNA repair and increase genomic instability
  6. Attacking the flank: Targeting new pathways in SBMA
  7. Guidelines for the use and interpretation of assays for monitoring autophagy
  8. Testosterone treatment fails to accelerate disease in a transgenic mouse model of spinal and bulbar muscular atrophy
  9. SIRT1 modulates aggregation and toxicity through deacetylation of the androgen receptor in cell models of SBMA
  10. An interdomain interaction of the androgen receptor is required for its aggregation and toxicity in spinal and bulbar muscular atrophy
  11. Autophagy and access: Understanding the role of androgen receptor subcellular localization in SBMA
  12. FOXO3a is broadly neuroprotective in vitro and in vivo against insults implicated in motor neuron diseases
  13. Cytoplasmic retention of polyglutamine-expanded androgen receptor ameliorates disease via autophagy in a mouse model of spinal and bulbar muscular atrophy
  14. Cyclin D1 repressor domain mediates proliferation and survival in prostate cancer
  15. ASC-J9 ameliorates spinal and bulbar muscular atrophy phenotype via degradation of androgen receptor
  16. Soluble androgen receptor oligomers underlie pathology in a mouse model of spinobulbar muscular atrophy
  17. Animal models of Kennedy disease
  18. Castration restores function and neurofilament alterations of aged symptomatic males in a transgenic mouse model of spinal and bulbar muscular atrophy
  19. Transglutaminase potentiates ligand-dependent proteasome dysfunction induced by polyglutamine-expanded androgen receptor
  20. Ligand promotes intranuclear inclusions in a novel cell model of spinal and bulbar muscular atrophy
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