16D0 Casimiro, Mathew - Thomas Jefferson University - Thomas Jefferson University
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Mathew C. Casimiro, PhD

Contact Dr. Casimiro

Kimmel Cancer Center
233 S. Tenth Street, Suite 1035
Philadelphia, PA 19107

(215) 503-9350
(215) 923-4498 fax

Most Recent Peer-reviewed Publications

  1. Overview of cyclins D1 function in cancer and the CDK inhibitor landscape: Past and present
  2. Identification of a cyclin D1 network in prostate cancer that antagonizes epithelial-mesenchymal restraint
  3. Cyclin D1 induction of dicer governs microRNA processing and expression in breast cancer
  4. Cyclin D1 determines estrogen signaling in the mammary gland in vivo
  5. Erratum: ChIP sequencing of cyclin D1 reveals a transcriptional role in chromosomal instability in mice (J Clin Invest. (2013) 123:5 (2332) 10.1172/JCI70042)
  6. Novel oncogene-induced metastatic prostate cancer cell lines define human prostate cancer progression signatures
  7. Cyclins and cell cycle control in cancer and disease
  8. Mitochondrial fission induces glycolytic reprogramming in cancer-associated myofibroblasts, driving stromal lactate production, and early tumor growth
  9. Cyclin D1 induces chromosomal instability
  10. ChIP sequencing of cyclin D1 reveals a transcriptional role in chromosomal instability in mice
  11. Mammary gland selective excision of c-Jun identifies its role in mRNA splicing
  12. Caveolin-1 overexpression enhances androgen-dependent growth and proliferation in the mouse prostate
  13. Examining the role of cyclin D1 in breast cancer
  14. Disruption of a Sirt1-dependent autophagy checkpoint in the prostate results in prostatic intraepithelial neoplasia lesion formation
  15. Cell fate determination factor Dachshund reprograms breast cancer stem cell function
  16. The canonical NF-κB pathway governs mammary tumorigenesis in transgenic mice and tumor stem cell expansion
  17. C-jun inhibits mammary apoptosis in vivo
  18. The autophagic tumor stroma model of cancer: Role of oxidative stress and ketone production in fueling tumor cell metabolism
  19. HIF1-alpha functions as a tumor promoter in cancer associated fibroblasts, and as a tumor suppressor in breast cancer cells: Autophagy drives compartment-specific oncogenesis
  20. Loss of stromal caveolin-1 leads to oxidative stress, mimics hypoxia and drives inflammation in the tumor microenvironment, conferring the "reverse Warb 015C urg effect": A transcriptional informatics analysis with validation
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