2238 Greenbaum, Linda E. - Thomas Jefferson University - Thomas Jefferson University
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Linda E. Greenbaum, MD

Contact Dr. Greenbaum

Kimmel Cancer Center
233 S. Tenth Street, Suite 519
Philadelphia, PA 19107

(215) 503-6345

Most Recent Peer-reviewed Publications

  1. Protein tyrosine phosphatase of liver regeneration-1 is required for normal timing of cell cycle progression during liver regeneration
  2. Ablation of Foxl1-Cre-labeled hepatic progenitor cells and their descendants impairs recovery of mice from liver injury
  3. Dynamic recruitment of microRNAs to their mRNA targets in the regenerating liver
  4. Robust cellular reprogramming occurs spontaneously during liver regeneration
  5. The role of paracrine signals during liver regeneration
  6. The ductal plate: A source of progenitors and hepatocytes in the adult liver
  7. Foxl1-Cre-marked adult hepatic progenitors have clonogenic and bilineage differentiation potential
  8. The role of stem cells in liver repair and fibrosis
  9. Tissue-specific regulation of mouse MicroRNA genes in endoderm-derived tissues
  10. Nuclear factor κB up-regulation of CCAAT/enhancer-binding protein β mediates hepatocyte resistance to tumor necrosis factor α toxicity
  11. From skin cells to hepatocytes: Advances in application of iPS cell technology
  12. Foxl1 promotes liver repair following cholestatic injury in mice
  13. Foxl1 is a marker of bipotential hepatic progenitor cells in mice
  14. Hedgehog signaling in biliary fibrosis
  15. Distinct proliferative and transcriptional effects of the D-type cyclins in vivo
  16. CCAAT/enhancer binding protein-β is a transcriptional regulator of peroxisome-proliferator-activated receptor-γ coactivator-1α in the regenerating liver
  17. C/EBPβ activates E2F-regulated genes in vi 057D vo via recruitment of the coactivator CREB-binding protein/p300
  18. Foxa2 integrates the transcriptional response of the hepatocyte to fasting
  19. Identification of transcriptional networks during liver regeneration
  20. Cell cycle regulation and hepatocarcinogenesis
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