2238 Greenbaum, Linda E. - Thomas Jefferson University - Thomas Jefferson University

Linda E. Greenbaum, MD

Contact Dr. Greenbaum

Kimmel Cancer Center
233 S. Tenth Street, Suite 519
Philadelphia, PA 19107

(215) 503-6345

Most Recent Peer-reviewed Publications

  1. A genetic screen reveals Foxa3 and TNFR1 as key regulators of liver repopulation
  2. Protein tyrosine phosphatase of liver regeneration-1 is required for normal timing of cell cycle progression during liver regeneration
  3. Ablation of Foxl1-Cre-labeled hepatic progenitor cells and their descendants impairs recovery of mice from liver injury
  4. Dynamic recruitment of microRNAs to their mRNA targets in the regenerating liver
  5. Robust cellular reprogramming occurs spontaneously during liver regeneration
  6. The role of paracrine signals during liver regeneration
  7. The ductal plate: A source of progenitors and hepatocytes in the adult liver
  8. Foxl1-Cre-marked adult hepatic progenitors have clonogenic and bilineage differentiation potential
  9. The role of stem cells in liver repair and fibrosis
  10. Tissue-specific regulation of mouse MicroRNA genes in endoderm-derived tissues
  11. Nuclear factor κB up-regulation of CCAAT/enhancer-binding protein β mediates hepatocyte resistance to tumor necrosis factor α toxicity
  12. From skin cells to hepatocytes: Advances in application of iPS cell technology
  13. Foxl1 promotes liver repair following cholestatic injury in mice
  14. Foxl1 is a marker of bipotential hepatic progenitor cells in mice
  15. Hedgehog signaling in biliary fibrosis
  16. Distinct proliferative and transcriptional effects of the D-type cyclins in vivo
  17. CCAAT/enhancer binding protein-β is a transcriptional regulator of peroxisome-proliferator-activated receptor-γ coac 059F tivator-1α in the regenerating liver
  18. C/EBPβ activates E2F-regulated genes in vivo via recruitment of the coactivator CREB-binding protein/p300
  19. Foxa2 integrates the transcriptional response of the hepatocyte to fasting
  20. Identification of transcriptional networks during liver regeneration