27EC Pestell, Richard G. - Thomas Jefferson University - Thomas Jefferson University

Richard G. Pestell, MD, PhD

Contact Dr. Pestell

233 South 10th Street
BLSB 908
Philadelphia, PA 19107

(215) 503-5649
(215) 503-9334 fax

Most Recent Peer-reviewed Publications

  1. Hepatocyte DACH1 Is Increased in Obesity via Nuclear Exclusion of HDAC4 and Promotes Hepatic Insulin Resistance
  2. Cancer stem cell metabolism
  3. Time-lapse video microscopy for assessment of EYFP-Parkin aggregation as a marker for cellular mitophagy
  4. Cancer metabolism: a therapeutic perspective
  5. A direct quantification method for measuring plasma MicroRNAs identified potential biomarkers for detecting metastatic breast cancer
  6. Cyclin D1 promotes androgen-dependent DNA damage repair in prostate cancer cells
  7. Breast cancer stem cell isolation
  8. The retinal determination gene network: From developmental regulator to cancer therapeutic target
  9. Cyclin D1 silencing suppresses tumorigenicity, impairs DNA double strand break repair and thus radiosensitizes androgen-independent prostate cancer cells to DNA damage
  10. The role of CD44 in epithelial–mesenchymal transition and cancer development
  11. EglN2 associates with the NRF1-PGC1α complex and controls mitochondrial function in breast cancer
  12. FXYD5 is a marker for poor prognosis and a potential driver for metastasis in ovarian carcinomas
  13. The endogenous cell-fate factor dachshund restrains prostate epithelial cell migration via repression of cytokine secretion via a CXCL signaling module
  14. Antibiotics for cancer therapy
  15. Endogenous Dach1 in cancer
  16. Emerging roles of peroxisome proliferator-activated receptor gamma in cancer
  17. Kinase independent oncogenic cyclin D1
  18. Mitochondrial mass and DNA repair in breast cancer stem cells
  19. Dissecting tumor metabolic heterogeneity: Telomerase and large cell size metabolically define a sub-population of stem-like, mitochondrial-rich, cancer cells
  20. Targeting tumor-initiating cells: Eliminating anabolic cancer stem cells with inhibitors of protein synthesis or by mimicking caloric restriction