2238 Lau, Wayne Bond - Thomas Jefferson University - Thomas Jefferson University
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Wayne Bond Lau, MD

Contact Dr. Lau

1020 Sansom Street
Thompson Building, Suite 239
Philadelphia, PA 19107

(215) 955-6844
(215) 923-6225 fax

Most Recent Peer-reviewed Publications

  1. Adiponectin inhibits tumor necrosis factor-α-induced vascular inflammatory response via caveolin-mediated ceramidase recruitment and activation
  2. Autoantibodies isolated from preeclamptic patients induce endothelial dysfunction via interaction with the angiotensin II AT1 receptor
  3. Dickkopf-3 attenuates pressure overload-induced cardiac remodelling
  4. BMP-2 overexpression augments vascular smooth muscle cell motility by upregulating myosin Va via Erk signaling
  5. Aging aggravates nitrate-mediated ROS/RNS changes
  6. Cardiomyocyte apoptosis after nonlethal traumatic injury
  7. Adiponectin inhibits oxidative/nitrative stress during myocardial ischemia and reperfusion via PKA signaling
  8. Differential regulation of TNF receptor 1 and receptor 2 in adiponectin expression following myocardial ischemia
  9. Inhibition of Fas-Associated Death Domain-Containing Protein (FADD) Protects against Myocardial Ischemia/Reperfusion Injury in a Heart Failure Mouse Model
  10. C1q/tumor necrosis factor-related protein-9, a novel adipocyte-derived cytokine, attenuates adverse remodeling in the ischemic mouse heart via protein kinase a activation
  11. Sevoflurane preconditioning attenuates myocardial ischemia/reperfusion injury via caveolin-3-dependent cyclooxygenase-2 inhibition
  12. Autoantibody against angiotensin AT1 receptor from preeclamptic patients enhances collagen-induced human platelet aggregation
  13. Ginsenoside Rd Attenuates Myocardial Ischemia/Reperfusion Injury via Akt/GSK-3β Signaling and Inhibition of the Mitochondria-Dependent Apoptotic Pathway
  14. Aging might augment reactive oxygen species (ROS) formation and affect reactive nitrogen species (RNS) level after myocardial ischemia/reperfusion in both humans and rats
  15. Hyperglycemia-Induced Protein Kinase C β2 Activation Induces Diastolic Cardiac Dysfunction in Diabetic Rats by Impairing Caveolin-3 Expression and Akt/eNOS Signaling
  16. Cardiomyocyte-expressed farnesoid-X-receptor is a novel apoptosis mediator and contributes to myocardial ischaemia/reperfusion injury
  17. Intermedin protects against myocardial ischemia-reperfusion injury in diabetic rats
  18. Increased expression of pigment epithelium-derived factor in aged mesenchymal stem cells impairs their therapeutic efficacy for attenuating myocardial infarction injury
  19. Lymphotoxin-α is a novel adiponectin expression suppressor following myocardial ischemia/reperfusion
  20. Tumor Necrosis Factor-α and Lymphotoxin-α Mediate Myocardial Ischemic Injury via TNF Receptor 1, but Are Cardioprotective When Activating TNF Receptor 2
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