0B68 Ma, Xin - Thomas Jefferson University - Thomas Jefferson University
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Xin-liang Ma, MD, PhD

Contact Dr. Ma

1020 Sansom Street
Thompson Building, Suite 239
Philadelphia, PA 19107

(215) 955-6844
(215) 923-6225 fax

Most Recent Peer-reviewed Publications

  1. Erratum: Long-term active immunization with a synthetic peptide corresponding to the second extracellular loop of β1-adrenoceptor induces both morphological and functional cardiomyopathic changes in rats (International Journal of Cardiology (2011) 149:1 (89-94))
  2. Adiponectin inhibits tumor necrosis factor-α-induced vascular inflammatory response via caveolin-mediated ceramidase recruitment and activation
  3. IRF8 suppresses pathological cardiac remodelling by inhibiting calcineurin signalling
  4. Autoantibodies isolated from preeclamptic patients induce endothelial dysfunction via interaction with the angiotensin II AT1 receptor
  5. Dickkopf-3 attenuates pressure overload-induced cardiac remodelling
  6. Adiponectin inhibits oxidative/nitrative stress during myocardial ischemia and reperfusion via PKA signaling
  7. Differential regulation of TNF receptor 1 and receptor 2 in adiponectin expression following myocardial ischemia
  8. Thioredoxin reductase was nitrated in the aging heart after myocardial ischemia/reperfusion
  9. C1q/tumor necrosis factor-related protein-9, a novel adipocyte-derived cytokine, attenuates adverse remodeling in the ischemic mouse heart via protein kinase a activation
  10. Sevoflurane preconditioning attenuates myocardial ischemia/reperfusion injury via caveolin-3-dependent cyclooxygenase-2 inhibition
  11. Creg promotes the proliferation of human umbilical vein endothelial cells through the ERK/cyclin E signaling pathway
  12. Autoantibody against angiotensin AT1 receptor from preeclamptic patients enhances collagen-induced human platelet aggregation
  13. Ginsenoside Rd Attenuates Myocardial Ischemia/Reperfusion Injury via Akt/GSK-3β Signaling and Inhibition of the Mitochondria-Dependent Apoptotic Pathway
  14. Hyperglycemia-Induced Protein Kinase C β2 Activation Induces Diastolic Cardiac Dysfunction in Diabetic Rats by Impairing Caveolin-3 Expression and Akt/eNOS Signaling
  15. Cardiomyocyte-expressed farnesoid-X-receptor is a novel apoptosis mediator and contributes to myocardial ischaemia/reperfusion injury
  16. Increased expression of pigment epithelium-derived factor in aged mesenchymal stem cells impairs their therapeutic efficacy for attenuating myocardial infarction injury
  17. Lymphotoxin-α is a novel adiponectin expression suppressor following myocardial ischemia/reperfusion
  18. Tumor Necrosis Factor-α and Lymphotoxin-α Mediate Myocardial Ischemic Injury via TNF Receptor 1, but Are Cardioprotective When Activating TNF Receptor 2
  19. GSK-3α is a central regulator of age-related pathologies in mice
  20. Inhibition of CTRP9, a novel and cardiac-abundantly expressed cell survival molecule, by TNFα-initiated oxidative signaling contributes to exacerbated cardiac injury in diabetic mice
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