0B68 Ma, Xin - Thomas Jefferson University - Thomas Jefferson University
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Xin-liang Ma, MD, PhD

Contact Dr. Ma

1020 Sansom Street
Thompson Building, Suite 239
Philadelphia, PA 19107

(215) 955-6844
(215) 923-6225 fax

Most Recent Peer-reviewed Publications

  1. Erratum: Long-term active immunization with a synthetic peptide corresponding to the second extracellular loop of β1-adrenoceptor induces both morphological and functional cardiomyopathic changes in rats (International Journal of Cardiology (2011) 149:1 (89-94))
  2. Adiponectin inhibits tumor necrosis factor-α-induced vascular inflammatory response via caveolin-mediated ceramidase recruitment and activation
  3. IRF8 suppresses pathological cardiac remodelling by inhibiting calcineurin signalling
  4. Autoantibodies isolated from preeclamptic patients induce endothelial dysfunction via interaction with the angiotensin II AT1 receptor
  5. Dickkopf-3 attenuates pressure overload-induced cardiac remodelling
  6. Activation of liver-X-receptor α but not liver-X-receptor β protects against myocardial ischemia/reperfusion injury
  7. Adiponectin inhibits oxidative/nitrative stress during myocardial ischemia and reperfusion via PKA signaling
  8. Differential regulation of TNF receptor 1 and receptor 2 in adiponectin expression following myocardial ischemia
  9. Thioredoxin reductase was nitrated in the aging heart after myocardial ischemia/reperfusion
  10. C1q/tumor necrosis factor-related protein-9, a novel adipocyte-derived cytokine, attenuates adverse remodeling in the ischemic mouse heart via protein kinase a activation
  11. Sevoflurane preconditioning attenuates myocardial ischemia/reperfusion injury via caveolin-3-dependent cyclooxygenase-2 inhibition
  12. Creg promotes the proliferation of human umbilical vein endothelial cells through the ERK/cyclin E signaling pathway
  13. Autoantibody against angiotensin AT1 receptor from preeclamptic patients enhances collagen-induced human platelet aggregation
  14. Ginsenoside Rd Attenuates Myocardial Ischemia/Reperfusion Injury via Akt/GSK-3β Signaling and Inhibition of the Mitochondria-Dependent Apoptotic Pathway
  15. Hyperglycemia-Induced Protein Kinase C β2 Activation Induces Diastolic Cardiac Dysfunction in Diabetic Rats by Impairing Caveolin-3 Expression and Akt/eNOS Signaling
  16. Cardiomyocyte-expressed farnesoid-X-receptor is a novel apoptosis mediator and contributes to myocardial ischaemia/reperfusion injury
  17. Increased expression of pigment epithelium-derived factor in aged mesenchymal stem cells impairs their therapeutic efficacy for attenuating myocardial infarction injury
  18. Lymphotoxin-α is a novel adiponectin expression suppressor following myocardial ischemia/reperfusion
  19. Tumor Necrosis Factor-α and Lymphotoxin-α Mediate Myocardial Ischemic Injury via TNF Receptor 1, but Are Cardioprotective When Activating TNF Receptor 2
  20. GSK-3α is a central regulator of age-related pathologies in mice
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