16D0 Schnell, Matthias J. - Thomas Jefferson University - Thomas Jefferson University

Matthias J. Schnell, PhD

Contact Dr. Schnell

233 South Tenth Street
Bluemle Life Sciences Building, Room 531
Philadelphia, PA 19107-5541

(215) 503-4634
(215) 503-5393 fax

Most Recent Peer-reviewed Publications

  1. Reverse genetics of Mononegavirales: How they work, new vaccines, and new cancer therapeutics
  2. Controlled viral glycoprotein expression as a safety feature in a bivalent rabies-ebola vaccine
  3. Rhabdovirus-based vaccine platforms against henipaviruses
  4. Safety and serological response to a matrix gene-deleted rabies virus-based vaccine vector in dogs
  5. Small-molecule probes targeting the viral PPxY-host nedd4 interface block egress of a broad range of RNA viruses
  6. Interspecies protein substitution to investigate the role of the lyssavirus glycoprotein
  7. Rabies virus (RV) glycoprotein expression levels are not critical for pathogenicity of RV
  8. Dendritic cells infected by recombinant rabies virus vaccine vector expressing HIV-1 Gag are immunogenic even in the presence of vector-specific immunity
  9. Rabies virus infe 1338 ction induces type I interferon production in an IPS-1 dependent manner while dendritic cell activation relies on IFNAR signaling.
  10. Induction of neutralizing antibody responses to anthrax protective antigen by using influenza virus vectors: Implications for disparate immune system priming pathways
  11. Rabies virus infection induces type I interferon production in an IPS-1 dependent manner while dendritic cell activation relies on IFNAR signaling
  12. Characterization of a single-cycle rabies virus-based vaccine vector
  13. A novel composite immunotoxin that suppresses rabies virus production by the infected cells
  14. Attenuation of rabies virulence: Takeover by the cytoplasmic domain of its envelope protein
  15. The cell biology of rabies virus: Using stealth to reach the brain
  16. Rabies virus-based vaccines elicit neutralizing antibodies, poly-functional CD8+ T cell, and protect rhesus macaques from AIDS-like disease after SIVmac251 challenge
  17. Replication-deficient rabies virus-based vaccines are safe and immunogenic in mice and nonhuman primates
  18. Intravenous inoculation of a bat-associated rabies virus causes lethal encephalopathy in mice through invasion of the brain via neurosecretory hypothalamic fibers
  19. Lineage-specific T-cell responses to Cancer mucosa antigen oppose systemic metastases without mucosal inflammatory disease
  20. Interferon-β expressed by a rabies virus-based HIV-1 vaccine vector serves as a molecular adjuvant and decreases pathogenicity