jxz108

Jianke Zhang, PhD

Contact Dr. Zhang

233 South Tenth Street
Bluemle Life Sciences Building, Room 731
Philadelphia, PA 19107

(215) 503-4559
(215) 503-4558 fax

Most Recent Peer-reviewed Publications

  1. Daxx plays a novel role in T cell survival but is dispensable in Fas-induced apoptosis
  2. MLKL and FADD Are Critical for Suppressing Progressive Lymphoproliferative Disease and Activating the NLRP3 Inflammasome
  3. A Robust Service Selection Method Based on Uncertain QoS
  4. Investigation on the Inversion of the Atmospheric Duct Using the Artificial Bee Colony Algorithm Based on Opposition-Based Learning
  5. Caspase-8 scaffolding function and MLKL regulate NLRP3 inflammasome activation downstream of TLR3
  6. Phosphorylation of FADD by the kinase CK1α promotes KRASG12D-induced lung cancer
  7. Sensor network deployment under distance uncertainty with robust optimization
  8. Inhibition of Fas-Associated Death Domain-Containing Protein (FADD) Protects against Myocardial Ischemia/Reperfusion Injury in a Heart Failure Mouse Model
  9. Anti-MS4a4B treatment abrogates MS4a4B-mediated protection in T cells and ameliorates experimental autoimmune encephalomyelitis
  10. Autophagosomal membrane serves as platform for intracellular death-inducing signaling complex (iDISC)-mediated caspase-8 activation and apoptosis
  11. RIP1-mediated regulation of lymphocyte survival and death responses
  12. RIP1-dependent and independent effects of necrostatin-1 in necrosis and T cell activation
  13. Epithelial cell death is an important contributor to oxidant-mediated acute lung injury
  14. Functional complementation between FADD and RIP1 in embryos and lymphocytes
  15. New robust optimization counterpart for linear optimization with uncertain data
  16. FADD deficiency impairs early hematopoiesis in the bone marrow
  17. Mammalian nitrilase 1 homologue Nit1 is a negative regulator in T cells
  18. The death domain of FADD is essential for embryogenesis, lymphocyte development, and proliferation
  19. A role for cFLIP in B cell proliferation and stress MAPK regulation
  20. The pyroptosome: A supramolecular assembly of ASC dimers mediating inflammatory cell death via caspase-1 activation