0B68 Zhang, Jianke - Thomas Jefferson University - Thomas Jefferson University

Jianke Zhang, PhD

Contact Dr. Zhang

233 South Tenth Street
Bluemle Life Sciences Building, Room 731
Philadelphia, PA 19107

(215) 503-4559
(215) 503-4558 fax

Most Recent Peer-reviewed Publications

  1. A Robust Service Selection Method Based on Uncertain QoS
  2. Caspase-8 scaffolding function and MLKL regulate NLRP3 inflammasome activation downstream of TLR3
  3. Sensor network deployment under distance uncertainty with robust optimization
  4. Inhibition of Fas-Associated Death Domain-Containing Protein (FADD) Protects against Myocardial Ischemia/Reperfusion Injury in a Heart Failure Mouse Model
  5. Anti-MS4a4B treatment abrogates MS4a4B-mediated protection in T cells and ameliorates experimental autoimmune encephalomyelitis
  6. Autophagosomal membrane serves as platform for intracellular death-inducing signaling complex (iDISC)-mediated caspase-8 activation and apoptosis
  7. RIP1-mediated regulation of lymphocyte survival and death responses
  8. RIP1-dependent and independent effects of necrostatin-1 in necrosis and T cell activation
  9. Epithelial cell death is an important contributor to oxidant-mediated acute lung injury
  10. Functional complementation between FADD and RIP1 in embryos and lymphocytes
  11. FADD deficiency impairs early hematopoiesis in the bone marrow
  12. New robust optimization counterpart for linear optimization with uncertain data
  13. Mammalian nitrilase 1 homologue Nit1 is a negative regulator in T cells
  14. The death domain of FADD is essential for embryogenesis, lymphocyte development, and proliferation
  15. A role for cFLIP in B cell proliferation and stress MAPK regulation
  16. The pyroptosome: A supramolecular assembly of ASC dimers mediating inflammatory cell death via caspase-1 activation
  17. Critical role for Daxx in regulating Mdm2
  18. The Fas-associated death domain protein is required in apoptosis and TLR-induced proliferative responses in B cells
  19. Structural requirements for signal-induced target binding of FADD determined by functional reconstitution of FADD deficiency
  20. Conditional Fas-associated death domain protein (FADD): GFP knockout mice reveal FADD is dispensable in thymic development but essential in peripheral T cell homeostasis