Rene Daniel, MD, PhD
1015 Walnut Street
Philadelphia, PA 19107
Philadelphia, PA 19107
Most Recent Peer-reviewed Publications
- Understanding lentiviral vector chromatin targeting: Working to reduce insertional mutagenic potential for gene therapy
- Stem cells and aging: A chicken-or-the-egg issue?
- Pyruvate kinase expression (PKM1 and PKM2) in cancer associated fibroblasts drives stromal nutrient production and tumor growth
- Proliferating cell nuclear antigen is required for loading of the SMCX/KMD5C histone demethylase onto chromatin
- Host Factors that 188D affect Provirus Stability and Silencing
- Up-regulation of HIV-1 transduction in nondividing cells by double-strand DNA break-inducing agents
- HIV-1 integration site preferences in pluripotent cells
- A role for the histone deacetylase HDAC4 in the life-cycle of HIV-1-based vectors
- A role for the Werner syndrome protein in epigenetic inactivation of the pluripotency factor Oct4
- Modification of integration site preferences of an HIV-1-based vector by expression of a novel synthetic protein
- HIV-1 Tat and AIDS-associated cancer: Targeting the cellular anti-cancer barrier?
- Integration site selection by retroviral vectors: Molecular mechanism and clinical consequences
- ATR-Chk2 signaling in p53 activation and DNA damage response during cisplatin-induced apoptosis (Journal of Biological Chemistry (2008) 283, (6572-6583))
- Abnormal cytokinesis after X-irradiation in tumor cells that override the G2 DNA damage checkpoint
- Evidence that the Nijmegen breakage syndrome protein, an early sensor of double-strand DNA breaks (DSB), is involved in HIV-1 post-integration repair by recruiting the ataxia telangiectasia-mutated kinase in a process similar to, but distinct from, cellular DSB repair
- ATR-Chk2 signaling in p53 activation and DNA damage response during cisplatin-induced apoptosis
- Pentoxifylline suppresses transduction by HIV-1-based vectors
- DNA repair in HIV-1 infection: A case for inhibitors of cellular co-factors?
- Following the path of the virus: the exploitation of host DNA repair mechanisms by retroviruses.
- Exogenous IL-7 induces Fas-mediated human neuronal apoptosis: Potential effects during human immunodeficiency virus type 1 infection