gfr001

Glenn Rall, PhD

Contact Dr. Rall

333 Cottman Avenue
Room 493
Philadelphia, PA 19111

(215) 728-3617

Most Recent Peer-reviewed Publications

  1. What Kaplan-Meier survival curves don't tell us about CNS disease
  2. CD4+ T cells require either B cells or CD8+ T cells to control spread and pathogenesis of a neurotropic infection
  3. Development of Complex Models to Study Co- and Polymicrobial Infections and Diseases
  4. Get It through Your Thick Head: Emerging Principles in Neuroimmunology and Neurovirology Redefine Central Nervous System "immune Privilege"
  5. Everything You Always Wanted to Know about Rabies Virus (But Were Afraid to Ask)
  6. Here I Am, Despite Myself
  7. Bst2/tetherin is induced in neurons by type I interferon and viral infection but is dispensable for protection against neurotropic viral challenge
  8. Homeostatic interferon expression in neurons is sufficient for early control of viral infection
  9. Interferon-induced RIP1/RIP3-mediated necrosis requires PKR and is licensed by FADD and caspases
  10. Extended JAK activation and delayed STAT1 dephosphorylation contribute to the distinct signaling profile of CNS neurons exposed to interferon-gamma
  11. Immune Clearance of Attenuated Rabies Virus Results in Neuronal Survival with Altered Gene Expression
  12. STAT1-independent control of a neurotropic measles virus challenge in primary neurons and infected mice
  13. CNS recruitment of CD8+ T lymphocytes specific for a peripheral virus infection triggers neuropathogenesis during polymicrobial challenge
  14. Blue moon neurovirology: The merits of studying rare CNS diseases of viral origin
  15. Lymphocytic choriomeningitis virus-induced mortality in mice is triggered by edema and brain herniation
  16. Making it to the synapse: Measles virus spread in and among neurons
  17. Altered levels of STAT1 and STAT3 influence the neuronal response to interferon gamma
  18. Neurokinin-1 enables measles virus trans-synaptic spread in neurons
  19. Receptor-independent spread of the measles virus is unique to neurons and involves fusion of synaptic membranes
  20. A compromise for co-PIs