16D0 Ciric, Bogoljub - Thomas Jefferson University - Thomas Jefferson University
Bogoljub Ciric, PhD

Bogoljub Ciric, PhD

Contact Dr.  Ciric

901 Walnut Street
Suite 400
Philadelphia, PA 19107

(215) 955-9425

Most Recent Peer-reviewed Publications

  1. IL-9 signaling affects central nervous system resident cells during inflammatory stimuli
  2. 1,25-Dihydroxyvitamin D3 enhances neural stem cell proliferation and oligodendrocyte differentiation
  3. Expression of GM-CSF in T cells is increased in multiple sclerosis and suppressed by IFN-β therapy
  4. Silencing IFN-γ binding/signaling in astrocytes versus microglia leads to opposite effects on central nervous system autoimmunity
  5. Emerging immunopharmacological targets in multiple sclerosis
  6. Astrocyte-derived lactosylceramide implicated in multiple sclerosis
  7. Tolerogenic dendritic cells produced by lentiviral-mediated CD40- and interleukin-23p19-specific shRNA can ameliorate experimental autoimmune encephalomyelitis by suppressing T helper type 17 cells
  8. Neurotrophin 3 transduction augments remyelinating and immunomodulatory capacity of neural stem cells
  9. Erratum: CNS-specific Thera 1291 py for Ongoing EAE by Silencing IL-17 Pathway in Astrocytes(Molecular Therapy)
  10. Committed Tc17 cells are phenotypically and functionally resistant to the effects of IL-27
  11. Immunotherapy using lipopolysaccharide-stimulated bone marrow-derived dendritic cells to treat experimental autoimmune encephalomyelitis
  12. GM-CSF in autoimmune inflammation of the central nervous system
  13. Role of Th17 cells in the pathogenesis of CNS inflammatory demyelination
  14. Intravenous transfer of apoptotic cell-treated dendritic cells leads to immune tolerance by blocking Th17 cell activity
  15. Immune tolerance induced by intravenous transfer of immature dendritic cells via up-regulating numbers of suppressive IL-10+ IFN-γ+-producing CD4+ T cells
  16. Accelerated and enhanced effect of CCR5-transduced bone marrow neural stem cells on autoimmune encephalomyelitis
  17. CNS-specific therapy for ongoing EAE by silencing IL-17 pathway in astrocytes
  18. IL-10 deficiency blocks the ability of LPS to regulate expression of tolerance-related molecules on dendritic cells
  19. Interleukin-10 plays a crucial role in suppression of experimental autoimmune encephalomyelitis by Bowman-Birk inhibitor
  20. Bowman-Birk inhibitor attenuates experimental autoimmune encephalomyelitis by delaying infiltration of inflammatory cells into the CNS