0B68 Rostami, Abdolmohamad - Thomas Jefferson University - Thomas Jefferson University
Abdolmohamad Rostami, MD, PhD

Abdolmohamad Rostami, MD, PhD

Contact Dr. Rostami

901 Walnut Street
Suite 400
Philadelphia, PA 19107

(215) 955-1234
(215) 955-1390 fax

Most Recent Peer-reviewed Publications

  1. Apoptotic cell-treated dendritic cells induce immune tolerance by specifically inhibiting development of CD4+ effector memory T cells
  2. 1,25-Dihydroxyvitamin D3 enhances neural stem cell proliferation and oligodendrocyte differentiation
  3. Analysis of 13 cell types reveals evidence for the expression of numerous novel primate- And tissue-specific microRNAs
  4. Expression of GM-CSF in T cells is increased in multiple sclerosis and suppressed by IFN-β therapy
  5. Silencing IFN-γ binding/signaling in astrocytes versus microglia leads to opposite effects on central nervous system autoimmunity
  6. c-kit plays a critical role in induction of intravenous tolerance in experimental autoimmune encephalomyelitis
  7. Mechanisms of immunological tolerance in central nervous system inflammatory demyelination
  8. Astrocyte-derived lactosylceramide implicated in multiple sclerosis
  9. Interferon regulatory factor (IRF) 3 is critical for the development of experimental autoimmune encephalomyelitis
  10. Tolerogenic dendritic cells produced by lentiviral-mediated CD40- and interleukin-23p19-specific shRNA can ameliorate experimental autoimmune encephalomyelitis by suppressing T helper type 17 cells
  11. Neurotrophin 3 transduction augments remyelinating and immunomodulatory capacity of neural stem cells
  12. DAB389IL-2 suppresses autoimmune inflammation in the CNS and inhibits T cell-mediated lysis of glial target cells
  13. Erratum: CNS-specific Therapy for Ongoing EAE by Silencing IL-17 Pathway in Astrocytes(Molecular Therapy)
  14. Immunotherapy using lipopolysaccharide-stimulated bone marrow-derived dendritic cells to treat experimental autoimmune encephalomyelitis
  15. Committed Tc17 cells are phenotypically and functionally resistant to the effects of IL-27
  16. GM-CSF in autoimmune inflammation of the central nervous system
  17. Role of Th17 cells in the pathogenesis of CNS inflammatory demyelination
  18. Different mechanisms of inflammation induced in virus and autoimmune-mediated models of multiple sclerosis in C57BL6 mice
  19. Anti-MS4a4B treatment abrogates MS4a4B-mediated protection in T cells and ameliorates experimental autoimmune encephalomyelitis
  20. Intravenous transfer of apoptotic cell-treated dendritic cells leads to immune tolerance by blocking Th17 cell activity
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