2238 Rostami, Abdolmohamad - Thomas Jefferson University - Thomas Jefferson University
Abdolmohamad Rostami, MD, PhD

Abdolmohamad Rostami, MD, PhD

Contact Dr. Rostami

901 Walnut Street
Suite 400
Philadelphia, PA 19107

(215) 955-1234
(215) 955-1390 fax

Most Recent Peer-reviewed Publications

  1. Interferon regulatory factor (IRF) 3 is critical for the development of experimental autoimmune encephalomyelitis
  2. Tolerogenic dendritic cells produced by lentiviral-mediated CD40- and interleukin-23p19-specific shRNA can ameliorate experimental autoimmune encephalomyelitis by suppressing T helper type 17 cells
  3. Neurotrophin 3 transduction augments remyelinating and immunomodulatory capacity of neural stem cells
  4. DAB389IL-2 suppresses autoimmune inflammation in the CNS and inhibits T cell-mediated lysis of glial target cells
  5. Role of Th17 cells in the pathogenesis of CNS inflammatory demyelination
  6. Different mechanisms of inflammation induced in virus and autoimmune-mediated models of multiple sclerosis in C57BL6 mice
  7. Anti-MS4a4B treatment abrogates MS4a4B-mediated protection in T cells and ameliorates experimental autoimmune encephalomyelitis
  8. Intravenous transfer of apoptotic cell-treated dendritic cells leads to immune tolerance by blocking Th17 cell activity
  9. Immune tolerance induced by intravenous transfer of immature dendritic cells via up-regulating numbers of suppressive IL-10+ IFN-γ+-producing CD4+ T cells
  10. Inhibitory effect of IL-17 on neural stem cell proliferation and neural cell differentiation
  11. Generation of large numbers of highly purified dendritic cells from bone marrow progenitor cells after co-culture with syngeneic murine splenocytes
  12. Independent and interdependent immunoregulatory effects of IL-27, IFN-β, and IL-10 in the suppression of human Th17 cells and murine experimental autoimmune encephalomyelitis
  13. Low dose zymosan ameliorates both chronic and relapsing experimental autoimmune encephalomyelitis
  14. Role of serine proteases in inflammation: Bowman-Birk protease inhibitor (BBI) as a potential therapy for autoimmune diseases
  15. Accelerated and enhanced effect of CCR5-transduced bone marrow neural stem cells on autoimmune encephalomyelitis
  16. TCR stimulation upregulates MS4a4B expression through induction of AP-1 transcription factor during T cell activation
  17. CNS-specific therapy for ongoing EAE by silencing IL-17 pathway in astrocytes
  18. Intravenous tolerance effectively overcomes enhanced pro-inflammatory responses and experimental autoimmune encephalomyelitis severity in the absence of IL-12 receptor signaling
  19. IL-10 deficiency blocks the ability of LPS to regulate expression of tolerance-related molecules on dendritic cells
  20. Interleukin-10 plays a crucial role in suppression of experimental autoimmune encephalomyelitis by Bowman-Birk inhibitor
0