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Kyunghee Koh, PhD

Contact Dr. Koh

900 Walnut Street
JHN 4th floor
Philadelphia, PA 19107

(215) 955-5905
(215) 503-4358 fax

Most Recent Peer-reviewed Publications

  1. Control of sleep by a network of cell cycle genes
  2. TARANIS Functions with Cyclin A and Cdk1 in a Novel Arousal Center to Control Sleep in Drosophila
  3. A neuron-glia interaction involving GABA transaminase contributes to sleep loss in sleepless mutants
  4. WIDE AWAKE mediates the circadian timing of sleep onset
  5. Regulation of synaptic development and function by the Drosophila PDZ protein Dyschronic
  6. Cell-specific fine-tuning of neuronal excitability by differential expression of modulator protein isoforms
  7. Genetic and Anatomical Basis of the Barrier Separating Wakefulness and Anesthetic-Induced Unresponsiveness
  8. Dyschronic, a Drosophila homolog of a deaf-blindness gene, regulates circadian output and slowpoke channels
  9. Post-translational regulation and nuclear entry of TIMELESS and PERIOD are affected in new timeless mutant
  10. SLEEPLESS, a Ly-6/neurotoxin family member, regulates the levels, localization and activity of Shaker
  11. The effects of caffeine on sleep in Drosophila require PKA activity, but not the adenosine receptor
  12. An isoform-specific mutant reveals a role of PDP1ε in the circadian oscillator
  13. The COP9 signalosome is required for light-dependent timeless degradation and Drosophila clock resetting
  14. Identification of SLEEPLESS, a sleep-promoting factor
  15. A genetic screen for sleep and circadian mutants reveals mechanisms underlying regulation of sleep in Drosophila
  16. Molecular analysis of sleep:wake cycles in Drosophila
  17. A Drosophila model for age-associated changes in sleep:wake cycles
  18. JETLAG resets the Drosophila circadian clock by promoting light-induced degradation of TIMELESS
  19. The nT1 translocation separates vulval regulatory elements from the egl-18 and elt-6 GATA factor genes
  20. Cell fates and fusion in the C. elegans vulval primordium are regulated by the EGL-18 and ELt-6 GATA factors - apparent direct targets of the LIN-39-Hox protein