0B68 Enomoto-Iwamoto, Motomi - Thomas Jefferson University - Thomas Jefferson University
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Motomi Enotomi-Iwamoto, PhD, DDS

Contact Dr. Enotomi-Iwamoto


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Most Recent Peer-reviewed Publications

  1. Distribution of slow-cycling cells in epiphyseal cartilage and requirement of β-catenin signaling for their maintenance in growth plate
  2. Epidermal growth factor receptor (EGFR) signaling regulates epiphyseal cartilage development through β-catenin-dependent and -independent pathways
  3. Epiphyseal abnormalities, trabecular bone loss and articular chondrocyte hypertrophy develop in the long bones of postnatal Ext1-deficient mice
  4. Toward regeneration of articular cartilage
  5. Heparan sulfate in skeletal development, growth, and pathology: The case of hereditary multiple exostoses
  6. Human umbilical cord blood-derived mesenchymal stem cells in the cultured rabbit intervertebral disc: A novel cell source for disc repair
  7. Loss of β-catenin induces multifocal periosteal chondroma-like masses in mice
  8. Erratum: Potent inhibition of heterotopic ossification by nuclear retinoic acid receptor-γ agonists (Nat. Med. (2012) 17 (454-460))
  9. 1349 Roles of Β-catenin signaling in phenotypic expression and proliferation of articular cartilage superficial zone cells
  10. The critical role of the epidermal growth factor receptor in endochondral ossification
  11. Intervertebral disc development is regulated by wnt/β-catenin signaling
  12. Potent inhibition of heterotopic ossification by nuclear retinoic acid receptor-γ agonists
  13. Synovial joint formation requires local Ext1 expression and heparan sulfate production in developing mouse embryo limbs and spine
  14. Endogenous retinoids in mammalian growth plate cartilage: Analysis and roles in matrix homeostasis and turnover
  15. Wnt/β-Catenin and retinoic acid receptor signaling pathways interact to regulate chondrocyte function and matrix turnover
  16. Transient activation of Wnt/β-catenin signaling induces abnormal growth plate closure and articular cartilage thickening in postnatal mice
  17. Retinoic acid receptors are required for skeletal growth, matrix homeostasis and growth plate function in postnatal mouse
  18. Wnt/β-catenin signaling stimulates matrix catabolic genes and activity in articular chondrocytes: Its possible role in joint degeneration (Laboratory Investigation (2008) 88, (264-274) DOI: 10.1038/labinvest.3700747)
  19. Small GTPase protein Rac-1 is activated with maturation and regulates cell morphology and function in chondrocytes
  20. A distinct cohort of progenitor cells participates in synovial joint and articular cartilage formation during mouse limb skeletogenesis
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