0B68 Enomoto-Iwamoto, Motomi - Thomas Jefferson University - Thomas Jefferson University

Motomi Enotomi-Iwamoto, PhD, DDS

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Most Recent Peer-reviewed Publications

  1. Resident mesenchymal progenitors of articular cartilage
  2. Mouse limb skeletal growth and synovial joint development are coordinately enhanced by Kartogenin
  3. Distribution of slow-cycling cells in epiphyseal cartilage and requirement of β-catenin signaling for their maintenance in growth plate
  4. Epidermal growth factor receptor (EGFR) signaling regulates epiphyseal cartilage development through β-catenin-dependent and -independent pathways
  5. Epiphyseal abnormalities, trabecular bone loss and articular chondrocyte hypertrophy develop in the long bones of postnatal Ext1-deficient mice
  6. Toward regeneration of articular cartilage
  7. Heparan sulfate in skeletal development, growth, and pathology: The case of hereditary multiple exostoses
  8. Human umbilical cord blood-derived mesenchymal stem cells in the cultured rabbit intervertebral disc: A novel cell source for disc repair
  9. Loss of β-catenin induces multifocal periosteal chondroma 05B4 -like masses in mice
  10. Erratum: Potent inhibition of heterotopic ossification by nuclear retinoic acid receptor-γ agonists (Nat. Med. (2012) 17 (454-460))
  11. Roles of Β-catenin signaling in phenotypic expression and proliferation of articular cartilage superficial zone cells
  12. The critical role of the epidermal growth factor receptor in endochondral ossification
  13. Intervertebral disc development is regulated by wnt/β-catenin signaling
  14. Potent inhibition of heterotopic ossification by nuclear retinoic acid receptor-γ agonists
  15. Synovial joint formation requires local Ext1 expression and heparan sulfate production in developing mouse embryo limbs and spine
  16. Endogenous retinoids in mammalian growth plate cartilage: Analysis and roles in matrix homeostasis and turnover
  17. Wnt/β-Catenin and retinoic acid receptor signaling pathways interact to regulate chondrocyte function and matrix turnover
  18. Transient activation of Wnt/β-catenin signaling induces abnormal growth plate closure and articular cartilage thickening in postnatal mice
  19. Retinoic acid receptors are required for skeletal growth, matrix homeostasis and growth plate function in postnatal mouse
  20. Wnt/β-catenin signaling stimulates ma 01CC trix catabolic genes and activity in articular chondrocytes: Its possible role in joint degeneration (Laboratory Investigation (2008) 88, (264-274) DOI: 10.1038/labinvest.3700747)