27EC Enomoto-Iwamoto, Motomi - Thomas Jefferson University - Thomas Jefferson University
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Motomi Enotomi-Iwamoto, PhD, DDS

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Most Recent Peer-reviewed Publications

  1. Resident mesenchymal progenitors of articular cartilage
  2. Tendon progenitor cells in injured tendons have strong chondrogenic potential: The cd105-negative subpopulation induces chondrogenic degeneration
  3. Mouse limb skeletal growth and synovial joint development are coordinately enhanced by Kartogenin
  4. Distribution of slow-cycling cells in epiphyseal cartilage and requirement of β-catenin signaling for their maintenance in growth plate
  5. Epidermal growth factor receptor (EGFR) signaling regulates epiphyseal cartilage development through β-catenin-dependent and -independent pathways
  6. Epiphyseal abnormalities, trabecular bone loss and articular chondrocyte hypertrophy develop in the long bones of postnatal Ext1-deficient mice
  7. Toward regeneration of articular cartilage
  8. Heparan sulfate in skeletal development, growth, and pathology: The case of hereditary multiple exostoses
  9. Human umbilical cord blood-derived mesenchymal stem cells in the cultured rabbit intervertebral disc: A novel cell source for disc repair
  10. Loss of β-catenin induces multifocal periosteal chondroma-like masses in mice
  11. Erratum: Potent inhibition of heterotopic ossification by nuclear retinoic acid receptor-γ agonists (Nat. Med. (2012) 17 (454-460))
  12. Roles of Β-catenin signaling in phenotypic expression and proliferation of articular cartilage superficial zone cells
  13. The critical role of the epidermal growth factor receptor in endochondral ossification
  14. Intervertebral disc development is regulated by wnt/β-catenin signaling
  15. Potent inhibition of heterotopic ossification by nuclear retinoic acid receptor-γ agonists
  16. Synovial joint formation requires local Ext1 expression and heparan sulfate production in developing mouse embryo limbs and spine
  17. Endogenous retinoids in mammalian growth plate cartilage: Analysis and roles in matrix homeostasis and turnover
  18. Wnt/β-Catenin and retinoic acid receptor signaling pathways interact to regulate chondrocyte function and matrix turnover
  19. Transient activation of Wnt/β-catenin signaling induces abnormal growth plate closure and articular cartilage thickening in postnatal mice
  20. Retinoic acid 0184 receptors are required for skeletal growth, matrix homeostasis and growth plate function in postnatal mouse
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