16D0 Pacifici, Maurizio - Thomas Jefferson University - Thomas Jefferson University
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Maurizio Pacifici, PhD

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Most Recent Peer-reviewed Publications

  1. The type 2 diabetes associated rs7903146 T allele within TCF7L2 is significantly under-represented in Hereditary Multiple Exostoses: Insights into pathogenesis
  2. Interactions of signaling proteins, growth factors and other proteins with heparan sulfate: Mechanisms and mysteries
  3. Heparanase stimulates chondrogenesis and is up-regulated in human ectopic cartilage: A mechanism possibly involved in hereditary multiple exostoses
  4. Genesis and morphogenesis of limb synovial joints and articular cartilage
  5. Introduction to the mini-review series "Articular cartilage: Biology, pathology and repair"
  6. TMJ degeneration in SAMP8 mice is accompanied by deranged ihh signaling
  7. Multiple hereditary exostoses (MHE): Elucidating the pathogenesis of a rare skeletal disorder through interdisciplinary research
  8. Lubricin is required for the structural integrity and post-natal maintenance of TMJ
  9. Mouse limb skeletal growth and synovial joint development are coordinately enh 1259 anced by Kartogenin
  10. Distribution of slow-cycling cells in epiphyseal cartilage and requirement of β-catenin signaling for their maintenance in growth plate
  11. Epiphyseal abnormalities, trabecular bone loss and articular chondrocyte hypertrophy develop in the long bones of postnatal Ext1-deficient mice
  12. Regulation of CCN2 gene expression and possible roles in developing tooth germs
  13. Heparan sulfate in skeletal development, growth, and pathology: The case of hereditary multiple exostoses
  14. Perichondrium phenotype and border function are regulated by Ext1 and heparan sulfate in developing long bones: A mechanism likely deranged in Hereditary Multiple Exostoses
  15. Loss of β-catenin induces multifocal periosteal chondroma-like masses in mice
  16. Distinct spatiotemporal roles of hedgehog signalling during chick and mouse cranial base and axial skeleton development
  17. Erratum: Potent inhibition of heterotopic ossification by nuclear retinoic acid receptor-γ agonists (Nat. Med. (2012) 17 (454-460))
  18. Muenke syndrome mutation, FgfR3 P244R, Causes TMJ Defects
  19. Roles of Β-catenin signaling in phenotypic expression and proliferation of articular cartilage superficial zone cells
  20. TMJ development and growth require primary cilia function
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