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Gyorgy Hajnoczky, MD, PhD

Contact Dr. Hajnoczky

1020 Locust Street
Alumni Hall, Room 527
Philadelphia, PA 19107

(215) 503-1427

Most Recent Peer-reviewed Publications

  1. Recessive mutations in MSTO1 cause mitochondrial dynamics impairment, leading to myopathy and ataxia
  2. MSTO1 is a cytoplasmic pro-mitochondrial fusion protein, whose mutation induces myopathy and ataxia in humans
  3. Intracellular Ca2+ Sensing: Its Role in Calcium Homeostasis and Signaling
  4. Tissue-Specific Mitochondrial Decoding of Cytoplasmic Ca2+ Signals Is Controlled by the Stoichiometry of MICU1/2 and MCU
  5. Increased mitochondrial nanotunneling activity, induced by calcium imbalance, affects intermitochondrial matrix exchanges
  6. Mitochondrial fusion dynamics is robust in the heart and depends on calcium oscillations and contractile activity
  7. Natural and induced mitochondrial phosphate carrier loss: Differential dependence of mitochondrial metabolism and dynamics and cell survival on the extent of depletion
  8. VDAC2-specific cellular functions and the underlying structure
  9. Subcellular ROS imaging methods: Relevance for the study of calcium signaling
  10. MICU1 regulation of mitochondrial Ca 2+ uptake dictates survival and tissue regeneration
  11. Redox Nanodomains Are Induced by and Control Calcium Signaling at the ER-Mitochondrial Interface
  12. Motifs of VDAC2 required for mitochondrial Bak import and tBid-induced apoptosis
  13. The mitochondrial phosphate carrier: Role in oxidative metabolism, calcium handling and mitochondrial disease
  14. Trans-mitochondrial coordination of cristae at regulated membrane junctions
  15. Mitochondrial ca2+ uptake by the voltage-dependent anion channel 2 regulates cardiac rhythmicity
  16. Erratum: MCUR1 is an essential component of mitochondrial Ca 2+ uptake that regulates cellular metabolism (Nat. Cell Biol. 14, 2012 (1336-1343))
  17. Mitochondrial Ca(2+) uptake by the voltage-dependent anion channel 2 regulates cardiac rhythmicity
  18. Essential versus accessory aspects of cell death: Recommendations of the NCCD 2015
  19. Loss of Miro1-directed mitochondrial movement results in a novel murine model for neuron disease
  20. Distribution and Apoptotic Function of Outer Membrane Proteins Depend on Mitochondrial Fusion