0B68 Hajnoczky, Gyorgy - Thomas Jefferson University - Thomas Jefferson University
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Gyorgy Hajnoczky, MD, PhD

Contact Dr. Hajnoczky

1020 Locust Street
Jefferson Alumni Hall, Suite 244
Philadelphia, PA 19107

(215) 503-1427

Most Recent Peer-reviewed Publications

  1. Isoform- and species-specific control of inositol 1,4,5-trisphosphate (IP3) receptors by reactive oxygen species
  2. Interactions between sarco-endoplasmic reticulum and mitochondria in cardiac and skeletal muscle-pivotal roles in Ca2+ and reactive oxygen species signaling
  3. MICU1 controls both the threshold and cooperative activation of the mitochondrial Ca2+ uniporter
  4. Erratum: MCUR1 is an essential component of mitochondrial Ca 2+uptake that regulates cellular metabolism (Nature Cell Biology (2012) 14 (1336-1343 ))
  5. MCUR1 is an essential component of mitochondrial Ca2+ uptake that regulates cellular metabolism
  6. Switch from ER-mitochondrial to SR-mitochondrial calcium coupling during muscle differentiation
  7. Mitochondrial morphology and dynamics in hepatocytes from normal and ethanol-fed rats
  8. Calcium transport across the inner mitochondrial membrane: Molecular mechanisms and pharmacology
  9. Bid-induced mitochondrial membrane permeabilization waves propagated by local reactive oxygen species (ROS) signaling
  10. Alignment of sarcoplasmic reticulum-mitochondrial junctions with mitochondrial contact points
  11. Altered fusion dynamics underlie unique morphological changes in mitochondria during hypoxia-reoxygenation stress
  12. Activation of the mitochondrial permeability transition pore modulates Ca2+ responses to physiological stimuli in adult neurons
  13. Calcium signalling: Fishing out molecules of mitochondrial calcium transport
  14. Biophysical properties of mitochondrial fusion events in pancreatic β-cells and cardiac cells unravel potential control mechanisms of its selectivity
  15. Imaging Interorganelle Contacts and Local Calcium Dynamics at the ER-Mitochondrial Interface
  16. MTCH2/MIMP is a major facilitator of tBID recruitment to mitochondria
  17. VDAC2 is required for truncated BID-induced mitochondrial apoptosis by recruiting BAK to the mitochondria
  18. SR/ER-mitochondrial local communication: Calcium and ROS
  19. Fluorometric methods for detection of mitochondrial membrane permeabilization in apoptosis.
  20. Mitochondrial kiss-and-run: Interplay between mitochondrial motility and fusion-fission dyna 011D mics
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