2AB0 Eto, Masumi - Thomas Jefferson University - Thomas Jefferson University
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Masumi Eto, PhD

Contact Dr. Eto

1020 Locust Street
436 JAH
Philadelphia, PA 19107

(215) 503-7891
(215) 503-2073 fax

Most Recent Peer-reviewed Publications

  1. F-actin clustering and cell dysmotility induced by the pathological W148R missense mutation of filamin B at the actin-binding domain
  2. Reconstituted human myosin light chain phosphatase reveals distinct roles of two inhibitory phosphorylation sites of the regulatory subunit, MYPT1
  3. Nuclear localization of CPI-17, a protein phosphatase-1 inhibitor protein, affects histone H3 phosphorylation and corresponds to proliferation of cancer and smooth muscle cells
  4. Endogenous inhibitor proteins that connect Ser/Thr kinases and phosphatases in cell signaling
  5. Caffeine relaxes smooth muscle through actin depolymerization
  6. Reciprocal regulation controlling the expression of CPI-17, a specific inhibitor protein for the myosin light chain phosphatase in vascular smooth muscle cells
  7. Molecular mechanism of telokin-mediated disinhibition of myosin light chain phosphatase and cAMP/cGMP-induced relaxation of gastrointestinal smooth muscle
  8. Interleukin 6 mediates production of interleukin 10 in metastatic melanoma
  9. Effects of a fluorescent myosin light chain phosphatase inhibitor on prostate cancer cells
  10. Heat shock augments angiotensin II-induced vascular contraction through increased production of reactive oxygen species
  11. Regulation of cellular protein phosphatase-1 (PP1) by phosphorylation of the CPI-17 family, C-kinase-activated PP1 Inhibitors
  12. Solution structure of the inhibitory phosphorylation domain of myosin phosphatase targeting subunit 1
  13. ROCK mediates phorbol ester-induced apoptosis in prostate cancer cells via p21 Cip1 up-regulation and JNK
  14. Phosphorylation-dependent autoinhibition of myosin light chain phosphatase accounts for Ca2+ sensitization force of smooth muscle contraction
  15. Expression of CPI-17 in smooth muscle during embryonic development and in neointimal lesion formation
  16. Nitric oxide-induced biphasic mechanism of vascular relaxation via dephosphorylation of CPI-17 and MYPT1
  17. Y27632, a Rho-activated kinase inhibitor, normalizes dysregulation in alpha1-adrenergic receptor-induced contraction of Lyon hypertensive rat artery smooth muscle
  18. Thromboxane A2-induced bi-directional regulation of cerebral arterial tone
  19. Phosphorylation-Induced Conformational Switching of CPI-17 Produces a Potent Myosin Phosphatase Inhibitor
  20. Assay for three-way interaction of protein phosphatase-1 (Glc7) with regulatory subunits plus phosphatase inhibitor-2
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