16D0 Siegman, Marion J. - Thomas Jefferson University - Thomas Jefferson University
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Marion J. Siegman, PhD

Contact Dr. Siegman

1020 Locust Street
Philadelphia, PA 19107

(215) 503-3975
(215) 503-2073 fax

Most Recent Peer-reviewed Publications

  1. The pathway for force transmission in the rat anococcygeus muscle: A tale of two tendons
  2. Structural limits on force production and shortening of smooth muscle
  3. Smooth muscle myocyte ultrastructure and contractility
  4. A force-activated kinase in a catch smooth muscle
  5. The N-terminal region of twitchin binds thick and thin contractile filaments: Redundant mechanisms of catch force maintenance
  6. Mechanism of catch force: Tethering of thick and thin filaments by twitchin
  7. Myosin cross-bridge kinetics and the mechanism of catch
  8. Catch force links and the low to high force transition of myosin
  9. Twitchin as a regulator of catch contraction in molluscan smooth muscle
  10. Obituary. Andrew P. Somlyo (1930-2004).
  11. Twitchin from molluscan catch muscle. Primary structure and relationship between site-specific phosphorylation and mechanical function
  12. Phosphorylation of molluscan twitchin by the cAMP-dependent protein kinase
  13. The myosin cross-bridge cycle and its control by twitchin phosphorylation in catch muscle
  14. Control of cross-bridge cycling by myosin light chain phosphorylation in mammalian smooth muscle
  15. Regulation of catch muscle by twitchin phosphorylation: Effects on force, ATPase, and shortening
  16. Phosphorylation of a twitchin-related protein controls catch and calcium sensitivity of force production in invertebrate smooth muscle
  17. Phosphorylation of a high molecular weight (-600 kDa) protein regulates catch in invertebrate smooth muscle
  18. Hypertrophy of colonic smooth muscle: Contractile proteins, shortening velocity, and regulation
  19. Hypertrophy of colonic smooth muscle: Structural remodeling, chemical composition, and force output
  20. Thiophosphorylation of the 130-kDa subunit is associated with a decreased activity of myosin light chain phosphatase in α-toxin-permeabilized smooth muscle
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