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Gyorgy Hajnoczky, MD, PhD

Contact Dr. Hajnoczky

1020 Locust Street
Alumni Hall, Room 527
Philadelphia, PA 19107

(215) 503-1427

Most Recent Peer-reviewed Publications

  1. Dysregulation of RyR Calcium Channel Causes the Onset of Mitochondrial Retrograde Signaling
  2. IP3 receptor isoforms differently regulate ER-mitochondrial contacts and local calcium transfer
  3. Coming together to define membrane contact sites
  4. Mitochondrial calcium exchange links metabolism with the epigenome to control cellular differentiation
  5. Dysregulation of Mitochondrial Ca2+ Uptake and Sarcolemma Repair Underlie Muscle Weakness and Wasting in Patients and Mice Lacking MICU1
  6. Redox regulation of ER and mitochondrial Ca 2+ signaling in cell survival and death
  7. Perturbed mitochondria-ER contacts in live neurons that model the amyloid pathology of Alzheimer's disease
  8. MICU1 Interacts with the D-Ring of the MCU Pore to Control Its Ca2+ Flux and Sensitivity to Ru360
  9. MICU1 Confers Protection from MCU-Dependent Manganese Toxicity
  10. Mitochondrial fusion and Bid-mediated mitochondrial apoptosis are perturbed by alcohol with distinct dependence on its metabolism
  11. Mitochondrial dynamics in adaptive and maladaptive cellular stress responses
  12. Endoplasmic Reticulum–Mitochondrial Contactology: Structure and Signaling Functions
  13. Molecular mechanisms of cell death: Recommendations of the Nomenclature Committee on Cell Death 2018
  14. Redox regulation of type-I inositol trisphosphate receptors in intact mammalian cells
  15. ROS Control Mitochondrial Motility through p38 and the Motor Adaptor Miro/Trak
  16. Mitochondrial Nanotunnels
  17. Recessive mutations in MSTO1 cause mitochondrial dynamics impairment, leading to myopathy and ataxia
  18. MSTO1 is a cytoplasmic pro-mitochondrial fusion protein, whose mutation induces myopathy and ataxia in humans
  19. Intracellular Ca2+ Sensing: Its Role in Calcium Homeostasis and Signaling
  20. Tissue-Specific Mitochondrial Decoding of Cytoplasmic Ca2+ Signals Is Controlled by the Stoichiometry of MICU1/2 and MCU