Ankita Srivastava, PhD

Research Assistant Professor

Contact Information

Ankita Srivastava, PhD

1020 Locust Street
314C Jefferson Alumni Hall
Philadelphia, PA 19107

Email Ankita Srivastava

215-955-0576

Research Assistant Professor

Education

Postdoctoral Fellow, Thomas Jefferson University, Philadelphia, PA -  2023
PhD, National Center for Cell Science, Savitribai Phule University, Pune, Maharashtra, India -  2018
MS, School of Biotechnology, Banaras Hindu University, Varanasi, Uttar Pradesh, India -  2011

Publications

Awards

  • 2024 Emerging Technologies Grant Award, Azenta Life Sciences
  • 2024 American Physiological Society APSselect paper in Physiological Genomics
  • 2022 Outstanding Poster Presentation Award, Postdoc Research Symposium, Thomas Jefferson University
  • 2022 Best Postdoc Mentor Award, Jefferson Postdoc Research Symposium, Thomas Jefferson University
  • 2013 Senior Research Fellowship, Council of Scientific and Industrial Research, India
  • 2010 Jawaharlal Nehru University Biotechnology Fellowship for MSc Program, India
  • 2010 Junior Research Fellowship, Council of Scientific and Industrial Research, India 

Research & Clinical Interests

Translational Pathobiology through Omics & AI-Driven Histopathology

We pursue translational research in end-stage liver disease, liver transplantation, and prostate cancer. A key goal is to uncover disease mechanisms and improve diagnosis, prognosis, and clinical decision-making by translating high-resolution molecular and histopathological data into actionable patient-specific insights. Our interdisciplinary approach integrates spatial transcriptomics, histopathology, AI-based image analysis, and machine learning towards advancing precision medicine. We are pursuing collaborative projects on:

  • integrated pathomics and transcriptomics towards developing therapy for alcohol- associated liver disease
  • enhancing liver transplantation by providing data-driven prognostic tools  for increased utilization of donor livers
  • deciphering morpho-molecular mechanisms driving tumor aggressiveness in prostate cancer

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